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As is well known, this disease was first described by the English physician James Parkinson in his original work entitled An essay on the saking palsy (An essay on the saking palsy, 1817). In this brief essay Parkinson wrote of the presence of “an involuntary tremulous movement, with diminished muscular strength, in the parts not in action, and even when sustained, with a propensity to bend the trunk forward”. That work gave us a magnificent description, based on observation of the patients Parkinson studied, of the disease, primarily of motor aspects of the disease, but also of non-motor aspects. A wonderful example of the work of the best tool ever invented, the human mind, in the service of others.

Around this date, many events of various kinds have been organized by patient and family groups, institutions, scientific societies, etc., in a commendable effort to remember that the fight against the disease is a task that involves the whole of society in general.

Parkinson’s patient Dancing!

Since 1997 the World Health Organization (WHO) has defined April 11 as World Parkinson’s Day, coinciding with the anniversary of the birth of Dr. James Parkinson, British neurologist who in 1817 discovered this neurodegenerative disease.

When you receive a diagnosis of Parkinson’s the impact is felt in all aspects of your life. The lack of knowledge of the disease at a social level, or the misconception that exists about it, leads you to have many doubts when communicating the diagnosis to those around you.

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1 – It is caused by a lack of dopamine in the body: Dopamine is a substance that transmits information necessary for us to perform movements normally. Lack of dopamine causes movement control to be altered, giving rise to typical motor symptoms such as resting tremor or rigidity. The loss (or degeneration) of neurons in the midbrain structure causes this lack of dopamine. At the onset of symptoms, about 60% of the dopamine-producing cells have already been lost, reducing dopamine levels by 80%.

The “bell of dreams” is already ringing at Quirónsalud

“These findings provide new evidence for the role of the gut in Parkinson’s disease and provide a model for studying the progression of the disease from the beginning,” explains Dr. Ted Dawson, director of the Johns Hopkins Institute for Cell Engineering and professor of neurology at the Johns Hopkins University School of Medicine.

Parkinson’s disease is caused by the concentration of an unfolded protein known as α-synuclein (alpha-synuclein), in brain neurons. As these proteins become more concentrated they produce neuronal death and leave as a sequel the loss of the substantia nigra and toxic aggregates called Lewy bodies. Neuronal death in the affected person impairs their ability to move, think and regulate their emotions.

Increasing evidence has implicated the brain-gut axis as the origin of Parkinson’s disease. The authors of the research wanted to test whether the unfolded α-synuclein proteins could spread via the vagus nerve, which runs like an electrical cable from the stomach and small intestine to the base of the brain.

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What is Gaucher disease?

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